JOVANA
Library Glossary Getting Started Three Levels Fields How it works Mission
Join the mission
Back to the library
Biology 1915

The Mechanism of Mendelian Heredity

T. H. Morgan, A. H. Sturtevant, H. J. Muller & C. B. Bridges

Genes are real points strung in a line along the chromosomes.

Choose your version
In depth · the introduction

Mendel had invisible “factors” that obeyed neat ratios. Morgan's flies showed those factors were real, physical things — strung in a line along the chromosomes, and you could even draw their map.

The big idea

Decades after Mendel, no one knew where his hereditary “factors” actually lived. Morgan's lab, breeding millions of fruit flies, pinned them to the chromosomes — the thread-like bodies cells carry. Each factor (we now say gene) rides on a particular chromosome at a particular spot.

The clinching trick was this: genes on the same chromosome tend to be inherited together (“linkage”), but sometimes chromosomes swap pieces (“crossing over”), which separates them. The closer two genes sit, the more rarely they get separated. So by counting how often two traits come apart in the offspring, you can measure the distance between their genes — and lay every gene out, in order, on a map.

How it came about

Thomas Hunt Morgan started out a skeptic — he doubted both Mendel's laws and the idea that chromosomes carried heredity. Then, around 1910, a single white-eyed male fly appeared in his cramped Columbia University “Fly Room,” and its odd pattern of inheritance pointed straight at the X-chromosome. The flies kept supplying answers, and Morgan changed his mind.

The breakthrough on mapping came from a 19-year-old undergraduate, Alfred Sturtevant. One night in 1911 he realised the crossing-over numbers could be turned into distances, and stayed up to draw the first gene map. Calvin Bridges nailed the cytological proof; Hermann Muller sharpened the theory. The four of them gathered the work into this 1915 book.

Why it mattered

This is the book that turned heredity into something you can measure and predict, not just describe. “Gene” became a place with coordinates. That single idea — map genes by how often traits recombine — built the first maps of our own genome and helped hunt down the genes behind diseases like cystic fibrosis, long before we could read DNA letter by letter. Morgan won the 1933 Nobel Prize for it.

A way to picture it

Imagine two charms threaded on the same string. Now and then the string is cut once and re-tied with its partner string, swapping whatever lies beyond the cut. If two charms sit far apart, a cut is likely to fall between them, so they often end up separated. If they sit almost touching, a cut rarely lands between them, so they nearly always stay together. Count how often each pair gets separated, and you've measured the gaps along the string — that is exactly how the gene map is drawn.

An interactive Drosophila X-chromosome: five sex-linked factors sit in a line on a ruler, and a pair of homologous chromosomes above shows a cross-over forming between the two genes you choose. Pick locus A and locus B and the readout reports their map distance and the chance the cross-over separates them — small for close genes, large for distant ones.

Where it sits

Mendel (1865) gave the abstract rules; Sutton and Boveri (1902–1903) guessed chromosomes carried them; this book turned the guess into a measured map. The thread runs onward to Avery (1944) and Watson–Crick (1953), who revealed the gene's chemistry as DNA — but Morgan's flies had already fixed the gene's address. Every genome map since, including the Human Genome Project, is a descendant of these fruit-fly charts.

The original document
Original source text

Preface — the problem of heredity

Morgan · Sturtevant · Muller · Bridges · The Mechanism of Mendelian Heredity · Henry Holt & Co., 1915 · Preface
From ancient times heredity has been looked upon as one of the central problems of biological philosophy.
The authors set out to show that the abstract Mendelian factors are carried by the chromosomes, and that the behaviour of the chromosomes in the germ-cells accounts, point for point, for the numerical results of breeding experiments.

Linkage & crossing over (Ch. III)

Chapter III · Linkage
When two factors enter a cross in the same chromosome they tend to be inherited together, rather than to assort independently as Mendel's law of free assortment would require.
The process of interchange between chromosomes is called crossing over; the tendency of factors to stay together is called linkage.
The strength of the linkage differs from pair to pair: yellow and white cross over in about 1 per cent. of cases, white and miniature in about 33 per cent., white and bar in about 44 per cent. Factors that are seldom separated must lie close together; factors often separated must lie far apart.

The linear arrangement of factors

Chapter III · The linear arrangement of factors shown by linkage relations
If the factors are arranged in a line along the chromosome, the chance of a break falling between any two of them is proportional to the distance that separates them. The percentage of crossing over therefore measures that distance, and from it the factors can be plotted, in order and to scale, upon a map.
Sturtevant's first such map (1913) placed five sex-linked factors of the fly upon a single line; the maps in this book extend the method, and the order on the map is the order in which the factors lie in the chromosome.

The factorial hypothesis (Ch. IX)

Chapter IX · The factorial hypothesis
The factor is treated as a definite thing, located at a definite point in a particular chromosome, transmitted by the chromosome from cell to cell and from parent to offspring, and capable, on rare occasions, of changing into a new form (a mutation) that is thereafter inherited in the same orderly way.
Columbia University · New York · 1915