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Insulin and Type 1 Diabetes: Replacing the Missing Hormone

Insulin is the cleanest example of replacement therapy — and one of the most dangerous drugs in everyday use. Learn what it does, how short- and long-acting preparations are stitched together to copy the body's daily rhythm, and why hypoglycaemia is the side effect that defines the whole therapy.

What insulin does, and why it must be injected

Insulin is the body's signal of plenty: released by the pancreas after a meal, it tells muscle and fat to pull glucose out of the blood and tells the liver to stop making more. In type 1 diabetes the cells that make it are destroyed, so glucose climbs to dangerous levels. The only treatment is to put insulin back — pure replacement of a missing hormone.

Insulin acts on a kinase-linked receptor on the cell surface, triggering glucose transporters to move to the membrane. Because it is a protein, swallowing it would simply mean digesting it — the gut breaks it into amino acids. So insulin is given by subcutaneous injection (or by pump), absorbed slowly from under the skin into the blood.

Copying the body's rhythm: basal and bolus

A healthy pancreas does two jobs: a steady low trickle all day and night (basal) and a sharp spike at each meal (bolus). Modern therapy copies this with two kinds of insulin engineered to have different half-lives. Rapid-acting insulins are taken at meals to cover the food; a long-acting insulin given once or twice daily provides the steady background. Mixing them lets the patient mimic a normal day.

Sketch of a day on basal-bolus insulin

Blood glucose target: roughly 4-7 mmol/L fasting

  Long-acting (basal): 1 dose ~ flat 24-h coverage
  Rapid-acting (bolus): 1 dose with each meal

Worked carb-counting bolus (illustrative):
  Insulin-to-carb ratio = 1 unit per 10 g carbohydrate
  Meal contains 60 g carbohydrate
  Meal bolus = 60 / 10 = 6 units rapid-acting

  Correction (illustrative):
  1 unit lowers glucose ~ 2 mmol/L
  Pre-meal glucose 12, target 6 -> excess 6 mmol/L
  Correction = 6 / 2 = 3 units added
  Total dose = 6 + 3 = 9 units
Illustrative only — real ratios are individualised and set by the diabetes team.

Hypoglycaemia: the side effect that defines the drug

Insulin's great danger is its own success: give too much, or eat too little after a dose, and blood glucose falls too low. Hypoglycaemia is the most important adverse drug reaction of insulin — it causes sweating, shaking, confusion, and, if severe, seizures or coma. It is a side effect that is simply the drug's main action taken too far, so it can never be fully designed out; it can only be managed by careful dosing and patient education.

  1. Recognise the warning signs early: sweating, hunger, trembling, a racing heart.
  2. Treat at once with fast sugar — glucose tablets, juice, sweets — then a longer-acting snack.
  3. For a collapsed or unconscious person, glucagon (by injection) or intravenous glucose is the rescue, never food by mouth.